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Disease-Modifying Treatments for Multiple Sclerosis Affect Measures of Cellular Immune Responses to EBNA-1 Peptides
Inhibition of pro-inflammatory signaling in human primary macrophages by enhancing arginase-2 via target site blockers
In silico prioritisation of microRNA-associated common variants in multiple sclerosis
Enhancing arginase 2 expression using target site blockers as a strategy to modulate macrophage phenotype
Mitochondrial arginase-2 is essential for IL-10 metabolic reprogramming of inflammatory macrophages
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