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Neurological disorders after severe pneumonia are associated with translocation of endogenous bacteria from the lung to the brain
From Molecules to Models: miRNAs and Advanced Human Platforms of Neurodegeneration and Repair in Multiple Sclerosis
Mitochondrial arginase-2 is essential for IL-10 metabolic reprogramming of inflammatory macrophages
Enhancing arginase 2 expression using target site blockers as a strategy to modulate macrophage phenotype
Inhibition of pro-inflammatory signaling in human primary macrophages by enhancing arginase-2 via target site blockers
Time-of-day control of mitochondria regulates NLRP3 inflammasome activation in macrophages
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